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[GetTopicListResponse(id=886621, encodeId=1b988866213d, content=這個課題本來是解釋:<a href='/topic/show?id=24448348527' target=_blank style='color:#2F92EE;'>#膽汁酸#</a>誘導<a href='/topic/show?id=3ed881343fa' target=_blank style='color:#2F92EE;'>#肝纖維化#</a>的機制的,這是臨床上常見的現(xiàn)象,但是機制并不清楚。過去認為可能是<a href='/topic/show?id=8c2120038ff' target=_blank style='color:#2F92EE;'>#上皮間質(zhì)化#</a>(EMT)導致的,當然炎癥信號通路肯定起關(guān)鍵作用,因此,<a href='/topic/show?id=7ba812e35f3' target=_blank style='color:#2F92EE;'>#NF-κB#</a>途徑是必然參與其中的,其它信號如TGF-beta, SMAD3等都會參與其中的。同時,肝纖維化和<a href='/topic/show?id=370d8126914' target=_blank style='color:#2F92EE;'>#肝癌#</a>時,<a href='/topic/show?id=4a0281923d' target=_blank style='color:#2F92EE;'>#GP73#</a>水平上升,它是<a href='/topic/show?id=36a8102336cb' target=_blank style='color:#2F92EE;'>#高爾基體#</a>上的重要蛋白,因此,將它們關(guān)聯(lián)起來,確實是一個比較巧妙的構(gòu)思!青年基金能想到這樣,相當不錯。, objectTitle=膽汁酸通過激活NF-κB途徑及破壞高爾基體結(jié)構(gòu)上調(diào)肝纖維化/肝硬化患者血清GP73水平的機制研究, objectType=nsfc, longId=905737, objectId=0318905e375e, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/sci/nsfc_show.do?id=0318905e375e, replyNumber=0, likeNumber=383, createdTime=2020-09-19, rootId=0, userName=lovetcm, userId=7a3710, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=0318905e375e, moduleTitle=膽汁酸通過激活NF-κB途徑及破壞高爾基體結(jié)構(gòu)上調(diào)肝纖維化/肝硬化患者血清GP73水平的機制研究, moduleType=nsfc, hrefUrl=http://ermita.cn/sci/nsfc_show.do?id=0318905e375e)], followStatus=false, userIsMember=false, type=1, lengthMark=1)]
梅斯話題小助手
2020-05-29